Metabolic Disease and Hepatocellular Carcinoma

van Thiel, David H.; Alwahsh, Salamah M; Ramadori, Giuliano

dc.contributor.author	van Thiel, David H.
dc.contributor.author	Alwahsh, Salamah M
dc.contributor.author	Ramadori, Giuliano
dc.date.accessioned	2021-05-04T08:02:51Z
dc.date.accessioned	2022-05-22T08:54:13Z
dc.date.available	2021-05-04T08:02:51Z
dc.date.available	2022-05-22T08:54:13Z
dc.date.issued	2016
dc.identifier.isbn	978-3-319-34214-6
dc.identifier.uri	https://www.springer.com/gp/book/9783319342122
dc.identifier.uri	http://localhost:8080/xmlui/handle/123456789/8236
dc.description	The pathophysiologic mechanisms recognized as inducing changes in the cell cycle and its regulation that enables carcinogenesis to occur are presented. More importantly, the mechanisms thought to be most important in each metabolic disorder that can terminate in the development of a hepatic cancer are identified. Often, more than one mechanism is involved and it is the sum mutation of these metabolic events with environmental hazards and exposures that enable cancer to develop or not in an individual with a metabolic disease having an association with hepatic cancer.	en_US
dc.language.iso	en	en_US
dc.publisher	Springer International Publishing Switzerland	en_US
dc.title	Metabolic Disease and Hepatocellular Carcinoma	en_US
dc.type	Book chapter	en_US